If you find that you can't fall out of bed every morning, you may be able to blame it in your genes, a new scientific research has been opened.
The study analyzed rich genetic information from the 23andMe DNA testing website and the British biobank to better understand what makes a "morning nut" or "night owl".
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"This study is important because it confirms that your morning or evening choice is at least to some extent dependent on genetic factors," said Michael Weedon, professor at Exeter Medical School University.
The study is the largest study of this kind – it covered the data of almost 700,000 people and found that significantly more genetic factors are related to people who sleep and wake up than they know before.
The researchers knew about the 24 genes associated with sleeping time, but the new study published on Wednesday Natural communication magazine, found an additional 327 role.
The analysis also showed that those who have a genetic tendency to sleep later have a higher risk of mental health problems, such as schizophrenia, although the authors warned that more work was needed to understand the link.
In the early stages of the study, the genes of people who reported themselves as "morning persons" or "evening persons" were analyzed.
Because these terms may mean different things to different people, the researchers tested a smaller number of participants who used activity trackers.
They looked at information from over 85,000 UK Biobank members who wore their hands to find objective data about their sleep patterns.
They discovered that the genes they identified could shift a person's natural awakening time up to 25 minutes, but there was no clear link between the genes and how long or how well people slept.
The study also looked at why some genes affect human sleep and wake up, finding differences in brain response to light and internal clock activity.
To test the long-standing theories of the relationship between sleep patterns and certain diseases, researchers also analyzed the correlation between "morning" and "evening" genes and various disorders.
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They discovered that genetic sleep and earlier awakening are associated with less risk of depression and schizophrenia and better welfare.
However, Weedon admitted that it is not obvious whether this link is a direct result for the morning person, or because the early parking is a lighter time in nine to five work environments.
Researchers plan to see if "genetically evening people have worse results if they are active in the morning compared to those whose genetics and activities are consistent," he said.
The study did not provide any evidence of a causal relationship between genes that affect sleep time and metabolic diseases, such as type 2 diabetes. But further research will look at individuals in these issues whose natural sleep slope does not match their lifestyle.
"For example, are individuals who are genetic evening people, but have been awakened early because work commitments are particularly susceptible to obesity and diabetes?"